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News Physiol Sci 11: 57-62, 1996;
1548-9213/96 $5.00
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News in Physiological Sciences, Vol 11, 57-62, Copyright © 1996 by International Union of Physiological Sciences

ARTICLES

Gastric Enterochromaffin-Like Cells and the Regulation of Acid Secretion

G Sachs and C Prinz

The interaction of gastrin, somatostatin, and other transmitters at the level of the histamine-containing enterochromaffin (ECL) cell is the major pathway determining rate of gastric acid secretion. Gastrin stimulates ECL cell elevation of [Ca2+]i, synthesis of histidine decarboxylase, histamine release, and cell growth by binding at a cholecystokinin-B receptor. Somatostatin inhibits gastrin-dependent elevation of [Ca2+]i and hence histamine release by binding to stomatostatin receptor 2 subtype.


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