Physiology Information on EB 2010
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

News Physiol Sci 15: 303-308, 2000;
1548-9213/00 $5.00
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Web of Science (9)
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Jones, S. P.
Right arrow Articles by Lefer, D. J.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Jones, S. P.
Right arrow Articles by Lefer, D. J.
News in Physiological Sciences, Vol. 15, No. 6, 303-308, December 2000
© 2000 Int. Union Physiol. Sci./Am. Physiol. Soc.

Myocardial Reperfusion Injury: Insights Gained from Gene-Targeted Mice

Steven P. Jones and David J. Lefer

S. P. Jones and D. J. Lefer are in the Department of Molecular and Cellular Physiology, Louisiana State University Health Sciences Center, 1501 Kings Highway, Shreveport, LA 71130.
Myocardial ischemia-reperfusion injury involves activation of multiple cell types, including leukocytes and endothelial cells. The pursuant inflammation involves diminished nitric oxide production, influx of neutrophils, and myocardial cell injury. Gene-targeted animals provide important clues about the progression of this inflammatory cascade.




This article has been cited by other articles:


Home page
 Am. J. Physiol. Heart Circ. Physiol.Home page
C. Kusmic, G. Lazzerini, F. Coceani, R. Barsacchi, A. L'Abbate, and G. Sambuceti
Paradoxical coronary microcirculatory constriction during ischemia: a synergic function for nitric oxide and endothelin
Am J Physiol Heart Circ Physiol, October 1, 2006; 291(4): H1814 - H1821.
[Abstract] [Full Text] [PDF]

Home page
 Exp PhysiolHome page
T. De Celle, J. P. Cleutjens, W. M. Blankesteijn, J. J. Debets, J. F. Smits, and B. J. Janssen
Long-term structural and functional consequences of cardiac ischaemia-reperfusion injury in vivo in mice
Exp Physiol, September 1, 2004; 89(5): 605 - 615.
[Abstract] [Full Text] [PDF]

Home page
 Arterioscler. Thromb. Vasc. Bio.Home page
C. Kupatt, C. Dessy, R. Hinkel, P. Raake, G. Daneau, C. Bouzin, P. Boekstegers, and O. Feron
Heat Shock Protein 90 Transfection Reduces Ischemia-Reperfusion-Induced Myocardial Dysfunction via Reciprocal Endothelial NO Synthase Serine 1177 Phosphorylation and Threonine 495 Dephosphorylation
Arterioscler Thromb Vasc Biol, August 1, 2004; 24(8): 1435 - 1441.
[Abstract] [Full Text] [PDF]

Home page
 Am. J. Physiol. Heart Circ. Physiol.Home page
S. P. Jones, S. D. Trocha, and D. J. Lefer
Cardioprotective actions of endogenous IL-10 are independent of iNOS
Am J Physiol Heart Circ Physiol, July 1, 2001; 281(1): H48 - H52.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Visit Other APS Journals Online