Physiology Cambridge Electronic Design Limited
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


News Physiol Sci 17: 51-55, 2002;
1548-9213/02 $5.00
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via ISI Web of Science (20)
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Obeso, J. A.
Right arrow Articles by Giménez-Amaya, J. M.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Obeso, J. A.
Right arrow Articles by Giménez-Amaya, J. M.
News in Physiological Sciences, Vol. 17, No. 2, 51-55, April 2002
© 2002 Int. Union Physiol. Sci./Am. Physiol. Soc.

The Basal Ganglia and Disorders of Movement: Pathophysiological Mechanisms

José A. Obeso1, María C. Rodríguez-Oroz1, Manuel Rodríguez4, Javier Arbizu2 and José M. Giménez-Amaya3

1 Departments of Neurology,
2 Nuclear Medicine, and
3 Anatomy, Clinica Universitaria and Medical School, University of Navarra, Pamplona 31008; and
4 Department of Physiology, Medical School, University of La Laguna, Tenerife 35042, Spain

The basal ganglia are part of a neuronal network organized in parallel circuits. The "motor circuit" is most relevant to the pathophysiology of movement. Abnormal increment or reduction in the inhibitory output activity of basal ganglia give rise, respectively, to poverty and slowness of movement (i.e., Parkinson's disease) or dyskinesias.




This article has been cited by other articles:


Home page
J. Neurophysiol.Home page
O. Ibanez-Sandoval, L. Carrillo-Reid, E. Galarraga, D. Tapia, E. Mendoza, J. C. Gomora, J. Aceves, and J. Bargas
Bursting in Substantia Nigra Pars Reticulata Neurons In Vitro: Possible Relevance for Parkinson Disease
J Neurophysiol, October 1, 2007; 98(4): 2311 - 2323.
[Abstract] [Full Text] [PDF]


Home page
Am. J. Physiol. Cell Physiol.Home page
I. Morales, J. G. Dopico, M. Sabate, T. Gonzalez-Hernandez, and M. Rodriguez
Substantia nigra osmoregulation: taurine and ATP involvement
Am J Physiol Cell Physiol, May 1, 2007; 292(5): C1934 - C1941.
[Abstract] [Full Text] [PDF]


Home page
Neurorehabil Neural RepairHome page
R. T. Jessop, C. Horowicz, and L. E. Dibble
Motor learning and Parkinson disease: refinement of movement velocity and endpoint excursion in a limits of stability balance task.
Neurorehabil Neural Repair, December 1, 2006; 20(4): 459 - 467.
[Abstract] [PDF]


Home page
JRSMHome page
E. A C Pereira and T. Z Aziz
Surgical insights into Parkinson's disease.
J R Soc Med, May 1, 2006; 99(5): 238 - 244.
[Abstract] [Full Text] [PDF]


Home page
Postgrad. Med. J.Home page
E A C Pereira and T Z Aziz
Parkinson's disease and primate research: past, present, and future.
Postgrad. Med. J., May 1, 2006; 82(967): 293 - 299.
[Abstract] [Full Text] [PDF]


Home page
Vet PatholHome page
C. J. Zeiss
Neuroanatomical Phenotyping in the Mouse: The Dopaminergic System
Vet. Pathol., November 1, 2005; 42(6): 753 - 773.
[Abstract] [Full Text] [PDF]


Home page
J. Neurosci.Home page
L. C. Parr-Brownlie and B. I. Hyland
Bradykinesia Induced by Dopamine D2 Receptor Blockade Is Associated with Reduced Motor Cortex Activity in the Rat
J. Neurosci., June 15, 2005; 25(24): 5700 - 5709.
[Abstract] [Full Text] [PDF]


Home page
Arch NeurolHome page
J. A. Vilensky and S. Gilman
Using Extirpations to Understand the Human Motor Cortex: Horsley, Foerster, and Bucy
Arch Neurol, March 1, 2003; 60(3): 446 - 451.
[Abstract] [Full Text] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Visit Other APS Journals Online