|
|
||||||||
1 Department of Physiology and Biophysics, University of Texas Medical Branch, Galveston 77555-0641; and
2 Neurobiology Section, School of Biological Sciences, The University of Texas, Austin, Texas 78712
Ca2+-induced endocytotic vesicles undergo protein-mediated interactions to restore a selectively permeable barrier and propagated action potentials in severed invertebrate giant axons. Similar barrier-restoration phenomena observed in cultured mammalian cells with transected neurites suggest that cellular/molecular mechanisms that repair plasmalemmal damage are phylogenetically conserved.
This article has been cited by other articles:
![]() |
H. Erez, G. Malkinson, M. Prager-Khoutorsky, C. I. De Zeeuw, C. C. Hoogenraad, and M. E. Spira Formation of microtubule-based traps controls the sorting and concentration of vesicles to restricted sites of regenerating neurons after axotomy J. Cell Biol., February 12, 2007; 176(4): 497 - 507. [Abstract] [Full Text] [PDF] |
||||
![]() |
C. D. McCaig, A. M. Rajnicek, B. Song, and M. Zhao Controlling Cell Behavior Electrically: Current Views and Future Potential Physiol Rev, July 1, 2005; 85(3): 943 - 978. [Abstract] [Full Text] [PDF] |
||||
![]() |
R. M. S. Weragoda, E. Ferrer, and E. T. Walters Memory-Like Alterations in Aplysia Axons after Nerve Injury or Localized Depolarization J. Neurosci., November 17, 2004; 24(46): 10393 - 10401. [Abstract] [Full Text] [PDF] |
||||
| HOME | HELP | FEEDBACK | SUBSCRIPTIONS | ARCHIVE | SEARCH | TABLE OF CONTENTS |
| Visit Other APS Journals Online |