Physiology Fuel your research with LabChart
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


Physiology 21: 380-387, 2006; doi:10.1152/physiol.00019.2006
1548-9213/06 $8.00
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via ISI Web of Science (18)
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Bers, D. M.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Bers, D. M.
Physiology, Vol. 21, No. 6, 380-387, December 2006
© 2006 Int. Union Physiol. Sci./Am. Physiol. Soc.

REVIEW

Altered Cardiac Myocyte Ca Regulation In Heart Failure

Donald M. Bers

Department of Physiology, Loyola University Chicago, Stritch School of Medicine, Maywood, Illinois dbers{at}lumc.edu

Ca is critical in both the electrical and mechanical properties of cardiac myocytes, and much is known about ionic currents and the normal excitation-contraction coupling process. In heart failure, there are significant alterations in how myocyte Ca is regulated, and these alterations are critical in dictating both contractile dysfunction and certain cardiac arrhythmias that are characteristic of heart failure.




This article has been cited by other articles:


Home page
J. Pharmacol. Exp. Ther.Home page
M. Rocchetti, M. Alemanni, G. Mostacciuolo, P. Barassi, C. Altomare, R. Chisci, R. Micheletti, P. Ferrari, and A. Zaza
Modulation of Sarcoplasmic Reticulum Function by PST2744 [Istaroxime; (E,Z)-3-((2-Aminoethoxy)imino) Androstane-6,17-dione Hydrochloride)] in a Pressure-Overload Heart Failure Model
J. Pharmacol. Exp. Ther., September 1, 2008; 326(3): 957 - 965.
[Abstract] [Full Text] [PDF]


Home page
Circ. Res.Home page
L. E. Vinge, P. W. Raake, and W. J. Koch
Gene Therapy in Heart Failure
Circ. Res., June 20, 2008; 102(12): 1458 - 1470.
[Abstract] [Full Text] [PDF]


Home page
J. Appl. Physiol.Home page
O. Cohen, H. Kanana, R. Zoizner, C. Gross, U. Meiri, M. D. Stern, G. Gerstenblith, and M. Horowitz
Altered Ca2+ handling and myofilament desensitization underlie cardiomyocyte performance in normothermic and hyperthermic heat-acclimated rat hearts
J Appl Physiol, July 1, 2007; 103(1): 266 - 275.
[Abstract] [Full Text] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Visit Other APS Journals Online
Copyright © 2006 by the Int. Union Physiol. Sci./Am. Physiol. Soc.