HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Full Text Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in Web of Science Similar articles in PubMed Alert me to new issues of the journal Download to citation manager Citing Articles Citing Articles via HighWire Citing Articles via Web of Science (3) Citing Articles via Google Scholar Google Scholar Articles by Fry, A. C. Articles by Karet, F. E. Search for Related Content PubMed PubMed Citation Articles by Fry, A. C. Articles by Karet, F. E.

REVIEW

Inherited Renal Acidoses

Andrew C. Fry and Fiona E. Karet

Department of Medical Genetics and Division of Renal Medicine, University of Cambridge, Cambridge Institute for Medical Research fek1000{at}cam.ac.uk

Inherited acidosis may result from a primary renal defect in acid-base handling, emphasizing the central role of the kidney in control of body pH; as a secondary phenomenon resulting from abnormal renal electrolyte handling; or from excess production of acid elsewhere in the body. Here, we review our current understanding of the inherited renal acidoses at a genetic and molecular level.

This article has been cited by other articles:

				I. Carboni, E. Andreucci, M. R. Caruso, R. Ciccone, O. Zuffardi, M. Genuardi, I. Pela, and S. Giglio Medullary sponge kidney associated with primary distal renal tubular acidosis and mutations of the H+-ATPase genes Nephrol. Dial. Transplant., September 1, 2009; 24(9): 2734 - 2738. [Abstract] [Full Text] [PDF]

				H. Onken, S. K. Parks, G. G. Goss, and D. F. Moffett Serotonin-induced high intracellular pH aids in alkali secretion in the anterior midgut of larval yellow fever mosquito Aedes aegypti L. J. Exp. Biol., August 15, 2009; 212(16): 2571 - 2578. [Abstract] [Full Text] [PDF]