Physiology Fuel your research with LabChart
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


Physiology 22: 202-211, 2007; doi:10.1152/physiol.00044.2006
1548-9213/07 $8.00
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Fry, A. C.
Right arrow Articles by Karet, F. E.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Fry, A. C.
Right arrow Articles by Karet, F. E.
Physiology, Vol. 22, No. 3, 202-211, June 2007
© 2007 Int. Union Physiol. Sci./Am. Physiol. Soc.

REVIEW

Inherited Renal Acidoses

Andrew C. Fry and Fiona E. Karet

Department of Medical Genetics and Division of Renal Medicine, University of Cambridge, Cambridge Institute for Medical Research fek1000{at}cam.ac.uk

Inherited acidosis may result from a primary renal defect in acid-base handling, emphasizing the central role of the kidney in control of body pH; as a secondary phenomenon resulting from abnormal renal electrolyte handling; or from excess production of acid elsewhere in the body. Here, we review our current understanding of the inherited renal acidoses at a genetic and molecular level.







HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Visit Other APS Journals Online
Copyright © 2007 by the Int. Union Physiol. Sci./Am. Physiol. Soc.