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Physiology 22: 252-260, 2007; doi:10.1152/physiol.00012.2007
1548-9213/07 $8.00
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Physiology, Vol. 22, No. 4, 252-260, August 2007
© 2007 Int. Union Physiol. Sci./Am. Physiol. Soc.

REVIEW

Microvascular Dysfunction in Obesity: A Potential Mechanism in the Pathogenesis of Obesity-Associated Insulin Resistance and Hypertension

Amy M. Jonk1, Alfons J. H. M. Houben1, Renate T. de Jongh2, Erik H. Serné2, Nicoloaas C. Schaper1 and Coen D. A. Stehouwer1

1 Department of Internal Medicine, University Hospital Maastricht, and Cardiovascular Research Institute Maastricht, Maastricht; and
2 Department of Internal Medicine, VU Medical Center, Amsterdam, The Netherlands, csteh{at}sint.azm.nl

Obesity is an important risk factor for insulin resistance and hypertension and plays a central role in the metabolic syndrome. Insight into the pathophysiology of this syndrome may lead to new treatments. This paper has reviewed the evidence for an important role for the microcirculation as a possible link between obesity, insulin resistance and hypertension.




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