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Physiology 24: 298-306, 2009; doi:10.1152/physiol.00022.2009
1548-9213/09 $8.00
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Physiology, Vol. 24, No. 5, 298-306, October 2009
© 2009 Int. Union Physiol. Sci./Am. Physiol. Soc.

REVIEW

Role of Extracellular Adenosine in Acute Lung Injury

Tobias Eckle1, Michael Koeppen1 and Holger K. Eltzschig1,2

1 Department of Anesthesiology, Mucosal Inflammation Program, University of Colorado Denver, Aurora, Colorado; and
2 Department of Anesthesiology and Intensive Care Medicine, Tübingen University Hospital, Tübingen, Germany, holger.eltzschig{at}ucdenver.edu

Acute lung injury (ALI) is a lung disease characterized by pulmonary edema and severe hypoxia. The past decade hosted a search for endogenous mechanisms controlling lung inflammation and pulmonary edema during ALI. As such, recent evidence indicates extracellular adenosine in orchestrating the resolution of pulmonary edema and inflammation during ALI.






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