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News in Physiological Sciences, Vol 5, 245-249, Copyright © 1990 by International Union of Physiological Sciences
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AP Rocchini
Data are presented to suggest that obesity hypertension is primarily related to increased sodium retention and a concomitant increase in cardiac output. Evidence is also summarized suggesting sodium retention is due to combined effects of hyperinsulinemia, hyperaldosteronism, and/or increased sympathetic nervous system activity.
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